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5,17 Endothelial cells (ECs), key mediators of vascular lesions, absorb excess glucose via insulin-independent pathways, such as glucose transporter 1-3 (GLUT1-3), resulting in elevated intracellular glucose levels

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Comparing with wild genotype (CC), the heterozygote (CT) and mutation homozygote (TT) lead to the decline of enzyme activity by about 34 and 75%, and increased thermolability in lymphocyte extracts 25